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Although most anabolic and androgenic effects are expressed by the androgen receptor, some anabolic steroids can function outside the androgen receptor, particularly in the brain. Anabolic steroids were present even in high concentrations in the brain of male marmosets injected with estrogen but these animals could not ovulate or reproduce due to the high levels of estradiol. The androgen receptor is the site of action of all anabolic steroids in mammals. In humans it seems that the anabolic effects are largely due to an increase in muscle protein synthesis and breakdown (the increase in muscle protein synthesis is what causes the anabolic effects, since protein is a key amino acid for muscle protein synthesis) but the anabolic effects of a variety of other anabolic steroids are primarily due to the estrogenic steroid. Animal experiments Many experiments examining the anabolic androgenic actions of various steroid preparations on male rodents have been done. The studies on rats that first gave the rats oral testosterone (which is metabolized to 5α-dihydrotestosterone and 5α-dihydrotestosterone- androstenediol, d- and d/o-androstanediol) demonstrated that 5α-diphrenate (or 5α-dihydro 5α-dihydroandrosterone) was an effective anabolic agent and testosterone was an effective estrogenic agent. In the rat's testes, testosterone was shown to reduce androgen uptake by reducing concentrations of androgens in the androgen-sensitive cells (the androgen-sensitive cells are the cells that synthesize androgens). One rat with testosterone supplementation also showed lower estradiol levels. The study on a human being revealed that a man with low testosterone level was not able to ovulate or reproduce as his testosterone levels are in the normal range. A study done on a group of healthy men at an institute of natural medicine confirmed that testosterone does not decrease the activity of the testes in normal and healthy men. However, testosterone may interfere with the functions of the androgen receptor. A study done on rats revealed that a dose of testosterone (100-200 mg) was not sufficient to produce a significant change in testosterone concentration in human males. The authors concluded that it would be impossible for testosterone to inhibit the functions of the androgen receptor. The authors concluded that it would be hard to know in humans the effect of androgens and that testosterone may interact with androgen receptors, and that there is no evidence to support the use of testosterone in the treatment of males with low testosterone levels. Another study on rats, on another subject, on a subject, on another and Similar articles:

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